Hereditary and acquired angioedema: Problems and progress: Proceedings of the third C1 esterase inhibitor deficiency workshop and beyond
Top Cited Papers
- 1 September 2004
- journal article
- Published by Elsevier in Journal of Allergy and Clinical Immunology
- Vol. 114 (3) , S51-S131
- https://doi.org/10.1016/j.jaci.2004.06.047
Abstract
No abstract availableKeywords
This publication has 105 references indexed in Scilit:
- Functionally active complement is present in human ovarian follicular fluid and can be activated by seminal plasmaClinical and Experimental Immunology, 1992
- A dysfunctional Cl inhibitor protein with a new reactive center mutation (Arg‐444→Leu)FEBS Letters, 1992
- Angioedema: Manifestations and managementJournal of the American Academy of Dermatology, 1991
- Complete nucleotide sequence of the gene for human C1 inhibitor with an unusually high density of Alu elementsEuropean Journal of Biochemistry, 1991
- Identification of a new P1 residue mutation (444Arg→Ser) in a dysfunctional C1 inhibitor protein contained in a type II hereditary angioedema plasmaFEBS Letters, 1990
- Absence of Anti-Human Immunodeficiency Virus Types 1 and 2 Seroconversion after the Treatment of Hemophilia A or von Willebrand's Disease with Pasteurized Factor VIII ConcentrateNew England Journal of Medicine, 1989
- Proteolytic inactivation of plasma C1- inhibitor in sepsis.Journal of Clinical Investigation, 1989
- Absence of Hepatitis after Treatment with a Pasteurized Factor VIII Concentrate in Patients with Hemophilia and No Previous TransfusionsNew England Journal of Medicine, 1987
- Interaction of 125I‐labelled complement subcomponents Cr and Cs with protease inhibitors in plasmaFEBS Letters, 1979
- Serum complement profiles in infants and childrenThe Journal of Pediatrics, 1975