Spacial isolation of protein kinase C activation in thrombin stimulated human platelets
- 1 October 1988
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 156 (1) , 148-156
- https://doi.org/10.1016/s0006-291x(88)80817-9
Abstract
No abstract availableThis publication has 12 references indexed in Scilit:
- A role for Gi in control of thrombin receptor-phospholipase C coupling in human platelets.Journal of Biological Chemistry, 1988
- The Na+H+ antiporter is not involved in potentiation of thrombin-induced responses by epinephrineBiochemical and Biophysical Research Communications, 1988
- A model for intracellular translocation of protein kinase C involving synergism between Ca2+ and phorbol estersNature, 1985
- Inositol 1,4,5-trisphosphate releases Ca2+ from a Ca2+-transporting membrane vesicle fraction derived from human platelets.Journal of Biological Chemistry, 1985
- Activation of phospholipase C is dissociated from arachidonate metabolism during platelet shape change induced by thrombin or platelet-activating factor. Epinephrine does not induce phospholipase C activation or platelet shape change.Journal of Biological Chemistry, 1984
- Inositol trisphosphate and diacylglycerol as second messengersBiochemical Journal, 1984
- The role of protein kinase C in cell surface signal transduction and tumour promotionNature, 1984
- Thrombin-induced phosphodiesteratic cleavage of phosphatidylinositol bisphosphate in human platelets.Journal of Biological Chemistry, 1983
- Production of diglyceride from phosphatidylinositol in activated human platelets.Journal of Clinical Investigation, 1979
- Inositol phospholipids and cell surface receptor functionBiochimica et Biophysica Acta (BBA) - Reviews on Biomembranes, 1975