PAH–DNA Adducts, Cigarette Smoking, GST Polymorphisms, and Breast Cancer Risk

This publication has 66 references indexed in Scilit:

• PAH-DNA adducts in a Chinese population: relationship to PAH exposure, smoking and polymorphisms of metabolic and DNA repair genes
  Biomarkers, 2008
• Quantitation of N-Acetyl-S-(9,10-dihydro-9-hydroxy-10-phenanthryl)-l-cysteine in Human Urine: Comparison with Glutathione-S-transferase Genotypes in Smokers
  Chemical Research in Toxicology, 2006
• Cumulative genetic defects in carcinogen metabolism may increase breast cancer risk (The Netherlands)
  Cancer Causes & Control, 2005
• Residential environmental exposures and other characteristics associated with detectable PAH-DNA adducts in peripheral mononuclear cells in a population-based sample of adult females
  Journal of Exposure Science & Environmental Epidemiology, 2005
• A cross-sectional study of polycyclic aromatic hydrocarbon-DNA adducts and polymorphism of glutathioneS-transferases among heavy smokers by race/ethnicity
  Biomarkers, 2003
• Polymorphisms of theGSTP1 andGSTM1 genes and PAH-DNA adducts in human mononuclear white blood cells
  Environmental and Molecular Mutagenesis, 2000
• Tobacco Smoke Carcinogens and Lung Cancer
  JNCI Journal of the National Cancer Institute, 1999
• Influence of erythrocyte glutathione S-transferase T1 on sister chromatid exchanges induced by diepoxybutane in cultured human lymphocytes
  Mutagenesis, 1996
• The impact of glutathione s‐transferase M1 and cytochrome P450 1A1 genotypes on white‐blood‐cell polycyclic aromatic hydrocarbon‐dna adduct levels in humans
  Molecular Carcinogenesis, 1995
• The Glut athione S-Transferase Supergene Family: Regulation of GST and the Contribution of the lsoenzymes to Cancer Chemoprotection and Drug Resistance Part I
  Critical Reviews in Biochemistry and Molecular Biology, 1995