Adenosine A2 receptor antagonist facilitates the reversal of long-term potentiation (depotentiation) of evoked postsynaptic potentials but inhibits that of population spikes in hippocampal CA1 neurons
- 14 December 1992
- journal article
- Published by Elsevier in Neuroscience Letters
- Vol. 148 (1-2) , 148-150
- https://doi.org/10.1016/0304-3940(92)90826-s
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology (02259105, 03255222)
This publication has 9 references indexed in Scilit:
- NMDA receptors - their role in long-term potentiationPublished by Elsevier ,2003
- Adenosine (A2) antagonist inhibits induction of long-term potentiation of evoked synaptic potentials but not of the population spike in hippocampal CA1 neuronsBiochemical and Biophysical Research Communications, 1991
- Intracellular cAMP regulates the response to NMDA in hippocampal neuronsNeuroReport, 1991
- Reversal of long-term potentiation (depotentiation) induced by tetanus stimulation of the input to CA1 neurons of guinea pig hippocampal slicesBrain Research, 1991
- Mechanisms of long-term potentiation: EPSP/spike dissociation, intradendritic recordings, and glutamate sensitivityJournal of Neuroscience, 1988
- The role of NMDA receptors in learning and memoryNature, 1987
- RELEASE OF ATP FROM A SYNAPTOSOMAL PREPARATION BY ELEVATED EXTRACELLULAR K+ AND BY VERATRIDINEJournal of Neurochemistry, 1978
- Long‐lasting potentiation of synaptic transmission in the dentate area of the unanaesthetized rabbit following stimulation of the perforant pathThe Journal of Physiology, 1973
- Long‐lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant pathThe Journal of Physiology, 1973