Phthalate ester-induced gubernacular lesions are associated with reduced insl3 gene expression in the fetal rat testis
Top Cited Papers
- 1 February 2004
- journal article
- Published by Elsevier in Toxicology Letters
- Vol. 146 (3) , 207-215
- https://doi.org/10.1016/j.toxlet.2003.09.012
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Quantitative Changes in Gene Expression in Fetal Rat Testes following Exposure to Di(n-butyl) PhthalateToxicological Sciences, 2003
- Mutations of the GREAT gene cause cryptorchidismHuman Molecular Genetics, 2002
- Incidence at birth and natural history of cryptorchidism: A study of 10,730 consecutive male infantsJournal of Endocrinological Investigation, 2002
- The Insulin-3 Gene: Lack of a Genetic Basis for Human CryptorchidismJournal of Urology, 2002
- The Overexpression of the Insl3 in Female Mice Causes Descent of the OvariesMolecular Endocrinology, 2002
- Effects of environmental antiandrogens on reproductive development in experimental animalsHuman Reproduction Update, 2001
- Perinatal Exposure to the Phthalates DEHP, BBP, and DINP, but Not DEP, DMP, or DOTP, Alters Sexual Differentiation of the Male RatToxicological Sciences, 2000
- Hormonal Control of Gubernaculum Development during Testis Descent: Gubernaculum Outgrowth in Vitro Requires Both Insulin-Like Factor and AndrogenEndocrinology, 2000
- PROXIMAL INSERTION OF GUBERNACULUM TESTIS IN NORMAL HUMAN FETUSES AND IN BOYS WITH CRYPTORCHIDISMJournal of Urology, 2000
- Involvement of Insulin-Like Factor 3 (Insl3) in Diethylstilbestrol-Induced CryptorchidismEndocrinology, 2000