Insulin-like Growth Factor-binding Protein-3 Modulates Expression of Bax and Bcl-2 and Potentiates p53-independent Radiation-induced Apoptosis in Human Breast Cancer Cells

This publication has 47 references indexed in Scilit:

• The IGF axis and programmed cell death
  Immunology & Cell Biology, 1999
• Development of Resistance to Insulin-like Growth Factor Binding Protein-3 in Transfected T47D Breast Cancer Cells
  Biochemical and Biophysical Research Communications, 1998
• Insulin-like Growth Factor Binding Protein-3 Induces Apoptosis in MCF7 Breast Cancer Cells
  Biochemical and Biophysical Research Communications, 1997
• Overexpression of the Bcl-2 Protein Increases the Half-life of p21Bax
  Journal of Biological Chemistry, 1995
• Insulin‐like growth factor‐binding protein enhancement of insulin‐like growth factor‐i (IGF‐I)–mediated DNA synthesis and IGF‐I binding in a human breast carcinoma cell line
  Journal of Cellular Physiology, 1994
• Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
  Cell, 1993
• p53 is required for radiation-induced apoptosis in mouse thymocytes
  Nature, 1993
• Insulin-like growth factor-I (IGF-I) and transforming growth factor- beta 1 release IGF-binding protein-3 from human fibroblasts by different mechanisms
  Endocrinology, 1992
• The insulin-like growth factor binding proteins (IGFBPs) in human breast cancer
  Breast Cancer Research and Treatment, 1992
• Growth hormone-dependent insulin-like growth factor (IGF) binding protein both inhibits and potentiates IGF-I-stimulated DNA synthesis in human skin fibroblasts
  Biochemical and Biophysical Research Communications, 1988