INFLIXIMAB DOWNREGULATES INTERFERON-γ PRODUCTION IN ACTIVATED GUT T-LYMPHOCYTES FROM PATIENTS WITH CROHN'S DISEASE
- 31 August 2001
- Vol. 15 (4) , 212-222
- https://doi.org/10.1006/cyto.2001.0919
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Infliximab for the Treatment of Fistulas in Patients with Crohn's DiseaseNew England Journal of Medicine, 1999
- Staphylococcal enterotoxin‐A directly stimulates signal transduction and interferon‐γ production in psoriatic T‐cell linesTissue Antigens, 1998
- Spontaneous secretion of interferon γ and interleukin 4 by human intraepithelial and lamina propria gut lymphocytesGut, 1998
- Immunomodulation of Crohn's disease using TNF-αneutralizing monoclonal antibodiesClinical Nutrition, 1997
- Common clonal chromosome aberrations in cytokine-dependent continuous human T-lymphocyte cell linesCancer Genetics and Cytogenetics, 1995
- Treatment of Crohn's disease with anti-tumor necrosis factor chimeric monoclonal antibody (cA2)Gastroenterology, 1995
- Chimeric anti-TNF-α monoclonal antibody cA2 binds recombinant transmembrane TNF-α and activates immune effector functionsCytokine, 1995
- The 26-kD transmembrane form of tumor necrosis factor alpha on activated CD4+ T cell clones provides a costimulatory signal for human B cell activation.The Journal of Experimental Medicine, 1993
- Spontaneous release of interferon gamma by intestinal lamina propria lymphocytes in Crohn's disease. Kinetics of in vitro response to interferon gamma inducers.Gut, 1991
- Triggered human mucosal T cells release tumour necrosis factor-alpha and interferon-gamma which kill human colonic epithelial cellsClinical and Experimental Immunology, 1991