Analysis of the 5′-flanking region of the β-amyloid precursor protein gene that contributes to increased promoter activity in differentiated neuronal cells
- 5 May 2000
- journal article
- Published by Elsevier in Molecular Brain Research
- Vol. 77 (2) , 185-198
- https://doi.org/10.1016/s0169-328x(00)00051-6
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Increased expression of amyloid precursor protein and amyloid precursor-like protein 2 during trophic factor withdrawal-induced death of neuronal PC12 cellsMolecular Brain Research, 1998
- APP Gene Promoter Constructs Are Preferentially Expressed in the CNS and Testis of Transgenic MiceBiochemical and Biophysical Research Communications, 1997
- Nerve Growth Factor and Ras Regulate β‐Amyloid Precursor Protein Gene Expression in PC12 CellsJournal of Neurochemistry, 1996
- Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor proteinNature, 1995
- Structural features of the 5′ upstream regulatory region of the gene encoding rat amyloid precursor proteinGene, 1993
- Nerve growth factor does not influence the expression of β amyloid precursor protein mRNA in rat brain: in vivo and in vitro studiesBrain Research, 1993
- Nerve growth factor-induced neuronal differentiation is accompanied by differential induction and localization of the amyloid precursor protein (APP) in PC12 cells and variant PC12S cellsMolecular Brain Research, 1993
- Increased Expression of β‐Amyloid Protein Precursor and Microtubule‐Associated Protein τ During the Differentiation of Murine Embryonal Carcinoma CellsJournal of Neurochemistry, 1992
- Basic fibroblast growth factor prevents death of lesioned cholinergic neurons in vivoNature, 1988
- In situ hybridization of nucleus basalis neurons shows increased beta-amyloid mRNA in Alzheimer disease.Proceedings of the National Academy of Sciences, 1988