Proconvulsant and ‘anxiogenic’ effects of n-butyl β carboline-3-carboxylate, an endogenous benzodiazepine binding inhibitor from brain
- 1 June 1988
- journal article
- research article
- Published by Elsevier in Pharmacology Biochemistry and Behavior
- Vol. 30 (2) , 331-336
- https://doi.org/10.1016/0091-3057(88)90463-7
Abstract
No abstract availableKeywords
This publication has 8 references indexed in Scilit:
- Anxiolytic and anxiogenic drug effects on exploratory activity in an elevated plus-maze: A novel test of anxiety in the ratPublished by Elsevier ,2002
- Acute stress induces an increase in rat cerebral cortex levels of n-butyl-β-carboline-3-carboxylate, an endogenous benzodiazepine binding inhibitorNeurochemistry International, 1987
- Validation of open : closed arm entries in an elevated plus-maze as a measure of anxiety in the ratJournal of Neuroscience Methods, 1985
- A brain octadecaneuropeptide generated by tryptic digestion of DBI (diazepam binding inhibitor) functions as a proconflict ligand of benzodiazepine recognition sitesNeuropharmacology, 1984
- A Benzodiazepine Receptor—Mediated Model of AnxietyArchives of General Psychiatry, 1984
- Searching for endogenous ligand(s) of central benzodiazepine receptorsNeurochemistry International, 1983
- Acute and chronic stress effects on open field activity in the rat: Implications for a model of depressionNeuroscience & Biobehavioral Reviews, 1981
- Benzodiazepine Receptor: Demonstration in the Central Nervous SystemScience, 1977