Vascular lesions in mice with a deficit in Fas-mediated apoptosis and their transfer
- 31 August 1996
- journal article
- Published by Elsevier in International Journal of Cardiology
- Vol. 54, S11-S20
- https://doi.org/10.1016/s0167-5273(96)88771-7
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
- Ministry of Health, Labour and Welfare
This publication has 13 references indexed in Scilit:
- A new germline VH gene encoding a mouse nephritogenic autoantibodyImmunogenetics, 1995
- CD95 (Fas)-dependent elimination of self-reactive B cells upon interaction with CD4+T cellsNature, 1995
- Sequence analysis of the germ-line VH gene corresponding to a nephritogenic antibody in MRL/lpr lupus miceClinical and Experimental Immunology, 1995
- Generalized lymphoproliferative disease in mice, caused by a point mutation in the fas ligandCell, 1994
- Fas and Fas Ligand: A Death Factor and Its ReceptorPublished by Elsevier ,1994
- Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosisNature, 1992
- Anti-DNA antibodies from autoimmune mice arise by clonal expansion and somatic mutation.The Journal of Experimental Medicine, 1990
- The Mac-2 antigen is a galactose-specific lectin that binds IgE.The Journal of Experimental Medicine, 1989
- The role of clonal selection and somatic mutation in autoimmunityNature, 1987
- B220: a B cell-specific member of the T200 glycoprotein familyNature, 1981