Genetic polymorphisms of toll-like receptor 9 influence the immune response to CpG and contribute to hyper-IgM in primary biliary cirrhosis
- 30 June 2005
- journal article
- Published by Elsevier in Journal of Autoimmunity
- Vol. 24 (4) , 347-352
- https://doi.org/10.1016/j.jaut.2005.03.002
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Bacteria and human autoimmunity: the case of primary biliary cirrhosisCurrent Opinion in Rheumatology, 2004
- Modulation of Toll‐like Receptor 9 Responses through Synthetic Immunostimulatory Motifs of DNAAnnals of the New York Academy of Sciences, 2003
- Primary biliary cirrhosisThe Lancet, 2003
- Toll-Like Receptor Signaling PathwaysScience, 2003
- Single-nucleotide polymorphisms in the Toll-like receptor 9 gene (TLR9): frequencies, pairwise linkage disequilibrium, and haplotypes in three U.S. ethnic groups and exploratory case–control disease association studiesGenomics, 2003
- CpG Motifs in Bacterial DNA and Their Immune EffectsAnnual Review of Immunology, 2002
- Detection of Antimitochondrial Autoantibodies in Immunofluorescent Ama–Negative Patients With Primary Biliary Cirrhosis Using Recombinant AutoantigensHepatology, 2001
- TLR4 mutations are associated with endotoxin hyporesponsiveness in humansNature Genetics, 2000
- Staging of chronic nonsuppurative destructive cholangitis (syndrome of primary biliary cirrhosis)Virchows Archiv, 1978
- Primary Biliary Cirrhosis Showing a High Titer of AutoantibodyNew England Journal of Medicine, 1958