ACTIVATION OF VASCULAR SMOOTH MUSCLE K+ CHANNELS BY ENDOTHELIUM‐DERIVED RELAXING FACTORS
- 27 February 1999
- journal article
- review article
- Published by Wiley in Clinical and Experimental Pharmacology and Physiology
- Vol. 26 (2) , 180-184
- https://doi.org/10.1046/j.1440-1681.1999.03006.x
Abstract
1. Endothelium‐derived relaxing factors (EDRF), including nitric oxide (NO), prostacyclin (PGI2) and an as yet uncharacterized endothelium‐derived hyperpolarizing factor (EDHF), are now recognized to...Keywords
This publication has 31 references indexed in Scilit:
- ENDOTHELIUM‐DERIVED HYPERPOLARIZING FACTOR(S): SPECIES AND TISSUE HETEROGENEITYClinical and Experimental Pharmacology and Physiology, 1999
- Roles of calcium‐activated and voltage‐gated delayed rectifier potassium channels in endothelium‐dependent vasorelaxation of the rabbit middle cerebral arteryBritish Journal of Pharmacology, 1998
- Involvement of voltage-dependent potassium channels in the EDHF-mediated relaxation of rat hepatic arteryBritish Journal of Pharmacology, 1997
- Characterization of the potassium channels involved in EDHF‐mediated relaxation in cerebral arteriesBritish Journal of Pharmacology, 1997
- Regulation of Potassium Channels in Coronary Arterial Smooth Muscle by Endothelium-Derived VasodilatorsHypertension, 1997
- Localization of the Kv1.5 K+ channel protein in explanted cardiac tissue.Journal of Clinical Investigation, 1995
- Transfer of the scorpion toxin receptor to an insensitive potassium channelNeuron, 1994
- Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscleNature, 1994
- Cloning of an apamin binding protein of vascular smooth muscleProtein Journal, 1994
- Hyperpolarization and relaxation of arterial smooth muscle caused by nitric oxide derived from the endotheliumNature, 1990