A single-strand specific lesion drives MMS-induced hyper-mutability at a double-strand break in yeast
- 5 August 2010
- journal article
- Published by Elsevier in DNA Repair
- Vol. 9 (8) , 914-921
- https://doi.org/10.1016/j.dnarep.2010.06.005
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- The Transition of Closely Opposed Lesions to Double-Strand Breaks during Long-Patch Base Excision Repair Is Prevented by the Coordinated Action of DNA Polymerase δ and Rad27/Fen1Molecular and Cellular Biology, 2009
- Bypass specialists operate togetherThe EMBO Journal, 2009
- Systems based mapping demonstrates that recovery from alkylation damage requires DNA repair, RNA processing, and translation associated networksGenomics, 2008
- Human AlkB Homolog 1 Is a Mitochondrial Protein That Demethylates 3-Methylcytosine in DNA and RNAJournal of Biological Chemistry, 2008
- Synthesis and Structure of Duplex DNA Containing the Genotoxic Nucleobase Lesion N7-MethylguanineJournal of the American Chemical Society, 2008
- Transcription increases methylmethane sulfonate-induced mutations in alkB strains of Escherichia coliDNA Repair, 2008
- Apn1 and Apn2 endonucleases prevent accumulation of repair-associated DNA breaks in budding yeast as revealed by direct chromosomal analysisNucleic Acids Research, 2008
- Mutational specificity and genetic control of replicative bypass of an abasic site in yeastProceedings of the National Academy of Sciences, 2008
- Evidence for mutation showersProceedings of the National Academy of Sciences, 2007
- Too Many Mutants with Multiple MutationsCritical Reviews in Biochemistry and Molecular Biology, 2007