p27 Is Essential for the Antiproliferative Action of 1,25-Dihydroxyvitamin D3 in Primary, but Not Immortalized, Mouse Embryonic Fibroblasts
Open Access
- 1 October 2002
- journal article
- Published by Elsevier
- Vol. 277 (40) , 37301-37306
- https://doi.org/10.1074/jbc.m204162200
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Antiproliferative Effect of 1 ,25-Dihydroxyvitamin D3 in Human Prostate Cancer Cell Line LNCaP Involves Reduction of Cyclin-Dependent Kinase 2 Activity and Persistent G1 AccumulationEndocrinology, 1998
- 1,25-Dihydroxyvitamin D3 Stimulates Expression and Translocation of Protein Kinase Cα and Cδ via a Nongenomic Mechanism and Rapidly Induces Phosphorylation of a 33-kDa Protein in Acute Promyelocytic NB4 CellsPublished by Elsevier ,1996
- A Syndrome of Multiorgan Hyperplasia with Features of Gigantism, Tumorigenesis, and Female Sterility in p27 -Deficient MiceCell, 1996
- Transcriptional activation of the Cdk inhibitor p21 by vitamin D3 leads to the induced differentiation of the myelomonocytic cell line U937.Genes & Development, 1996
- Mice Lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint controlCell, 1995
- Inhibitors of mammalian G1 cyclin-dependent kinases.Genes & Development, 1995
- Principles of CDK regulationNature, 1995
- Cdk inhibitors: on the threshold of checkpoints and developmentCurrent Opinion in Cell Biology, 1994
- Spontaneous Immortalization of Mouse Fibroblasts Involves Structural Changes in Senescence-Inducing Protein, MortalinBiochemical and Biophysical Research Communications, 1993
- The E2F transcription factor is a cellular target for the RB proteinCell, 1991