Cyclic AMP produces desensitization of prostacyclin and adenosine A2 receptors in hybrid cell lines but does not affect Gs function
- 1 March 1992
- journal article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
- Vol. 1134 (2) , 157-163
- https://doi.org/10.1016/0167-4889(92)90039-e
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- Prostaglandin E1-mediated, cyclic AMP-independent, down-regulation of Gs alpha in neuroblastoma x glioma hybrid cells.Journal of Biological Chemistry, 1990
- Cross-regulation between G-protein-mediated pathways. Stimulation of adenylyl cyclase increases expression of the inhibitory G-protein, Gi alpha 2.Journal of Biological Chemistry, 1990
- Role of phosphorylation in desensitization of the β-adrenoceptorTrends in Pharmacological Sciences, 1990
- Multiple pathways of rapid beta 2-adrenergic receptor desensitization. Delineation with specific inhibitors.Journal of Biological Chemistry, 1990
- Segregation of discrete Gsα‐mediated responses that accompany homologous or heterologous desensitization in two related somatic hybridsBritish Journal of Pharmacology, 1990
- A selective binding site for 3H-NECA that is not an adenosine A2 receptorBiochemical Pharmacology, 1989
- The effect of iloprost on the ADP-ribosylation of Gsα (the α-subunit of Gs)Biochemical Journal, 1989
- Prostacyclin analogues reduce ADP‐ribosylation of the α‐subunit of the regulatory Gs‐protein and diminish adenosine (A2) responsiveness of plateletsBritish Journal of Pharmacology, 1987
- A highly sensitive adenylate cyclase assayAnalytical Biochemistry, 1974
- A simple and sensitive saturation assay method for the measurement of adenosine 3′:5′-cyclic monophosphateBiochemical Journal, 1971