Parkin and synphilin-1 isoform expression changes in Lewy body diseases
- 1 June 2007
- journal article
- research article
- Published by Elsevier in Neurobiology of Disease
- Vol. 26 (3) , 681-687
- https://doi.org/10.1016/j.nbd.2007.03.007
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Low alpha-synuclein 126 mRNA levels in dementia with Lewy bodies and Alzheimer diseaseNeuroReport, 2006
- Synphilin-1A: An aggregation-prone isoform of synphilin-1 that causes neuronal death and is present in aggregates from α-synucleinopathy patientsProceedings of the National Academy of Sciences, 2006
- Parkin-mediated lysine 63-linked polyubiquitination: A link to protein inclusions formation in Parkinson's and other conformational diseases?Neurobiology of Aging, 2005
- Parkin Mediates Nonclassical, Proteasomal-Independent Ubiquitination of Synphilin-1: Implications for Lewy Body FormationJournal of Neuroscience, 2005
- Differential expression of α‐synuclein isoforms in dementia with Lewy bodiesNeuropathology and Applied Neurobiology, 2004
- The autosomal recessive juvenile Parkinson disease gene product, parkin, interacts with and ubiquitinates synaptotagmin XIHuman Molecular Genetics, 2003
- Synphilin-1 Is Developmentally Localized to Synaptic Terminals, and Its Association with Synaptic Vesicles Is Modulated by α-SynucleinJournal of Biological Chemistry, 2002
- Co-association of parkin and α-synucleinNeuroReport, 2001
- Ubiquitination of a New Form of α-Synuclein by Parkin from Human Brain: Implications for Parkinson's DiseaseScience, 2001
- Parkin immunoreactivity in the brain of human and non‐human primates: An immunohistochemical analysis in normal conditions and in Parkinsonian syndromesJournal of Comparative Neurology, 2001