A constitutively active and nuclear form of the MAP kinase ERK2 is sufficient for neurite outgrowth and cell transformation
- 1 October 1998
- journal article
- research article
- Published by Elsevier in Current Biology
- Vol. 8 (21) , 1141-1152
- https://doi.org/10.1016/s0960-9822(07)00485-x
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Activation Mechanism of the MAP Kinase ERK2 by Dual PhosphorylationCell, 1997
- Putidaredoxin Reductase-Putidaredoxin-Cytochrome P450cam Triple Fusion ProteinJournal of Biological Chemistry, 1996
- Characterization of the Bone Morphogenetic Protein-2 as a Neurotrophic FactorPublished by Elsevier ,1996
- Mutation of Position 52 in ERK2 Creates a Nonproductive Binding Mode for Adenosine 5‘-Triphosphate,Biochemistry, 1996
- PD 098059 Is a Specific Inhibitor of the Activation of Mitogen-activated Protein Kinase Kinase in Vitro and in VivoJournal of Biological Chemistry, 1995
- Deletion of a conserved juxtamembrane sequence in Trk abolishes NGF-promoted neuritogenesisNeuron, 1995
- Activity of the MAP kinase ERK2 is controlled by a flexible surface loopStructure, 1995
- Multiple ras functions can contribute to mammalian cell transformationCell, 1995
- The sevenmaker gain‐of‐function mutation in p42 MAP kinase leads to enhanced signalling and reduced sensitivity to dual specificity phosphatase actionFEBS Letters, 1994
- Transformation of Mammalian Cells by Constitutively Active MAP Kinase KinaseScience, 1994