Analysis of Message Expression in Single Neurons of Alzheimer’s Disease Brain
Open Access
- 28 February 1998
- journal article
- Published by Elsevier in Neurobiology of Aging
- Vol. 19 (1) , S99-S105
- https://doi.org/10.1016/s0197-4580(98)00044-x
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- Isolation of single immunohistochemically identified whole neuronal cell bodies from post-mortem human brain for simultaneous analysis of multiple gene expressionJournal of Neuroscience Methods, 1997
- Neurons bearing neurofibrillary tangles are responsible for selected synaptic deficits in Alzheimer's diseaseNeurobiology of Aging, 1995
- Gene expression and cellular content of cathepsin D in Alzheimer's disease brain: Evidence for early up-regulation of the endosomal-lysosomal systemNeuron, 1995
- Preliminary evidence: Decreased GAP-43 message in tangle-bearing neurons relative to adjacent tangle-free neurons in Alzheimer's disease parahippocampal gyrusNeurobiology of Aging, 1994
- Reduced GAP-43 message levels are associated with increased neurofibrillary tangle density in the frontal association cortex (area 9) in Alzheimer's diseaseNeurobiology of Aging, 1992
- Analysis of gene expression in single live neurons.Proceedings of the National Academy of Sciences, 1992
- Neuron numbers and dendritic extent in normal aging and Alzheimer's diseaseNeurobiology of Aging, 1987
- Molecular Properties of the Growth‐Associated Protein GAP‐43 (B‐50)Journal of Neurochemistry, 1987
- Quantitative evidence for selective dendritic growth in normal human aging but not in senile dementiaBrain Research, 1981
- Dendritic Growth in the Aged Human Brain and Failure of Growth in Senile DementiaScience, 1979