Conjugated linoleic acid decreases production of pro-inflammatory products in macrophages: evidence for a PPARγ-dependent mechanism
- 29 March 2002
- journal article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
- Vol. 1581 (3) , 89-99
- https://doi.org/10.1016/s1388-1981(02)00126-9
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Peroxisome proliferator-activated receptors in macrophage biology: friend or foe?Current Opinion in Lipidology, 2001
- DIFFERENT MECHANISMS IN INHIBITION OF RAT MACROPHAGE NITRIC OXIDE SYNTHASE EXPRESSION BY FK 506 AND CYCLOSPORIN AImmunopharmacology and Immunotoxicology, 2001
- Activation of PPARgamma may mediate a portion of the anticancer activity of conjugated linoleic acidMedical Hypotheses, 2000
- Conjugated linoleic acid activates peroxisome proliferator-activated receptor α and β subtypes but does not induce hepatic peroxisome proliferation in Sprague–Dawley ratsBiochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids, 1999
- Peroxisome proliferator-activated receptor-γ: a versatile metabolic regulatorAnnals of Medicine, 1999
- Terminal Differentiation of Human Breast Cancer through PPARγMolecular Cell, 1998
- Rel/NF-κB and IκB proteins: an overviewSeminars in Cancer Biology, 1997
- Regulation of adipocyte gene expression and differentiation by peroxisome proliferator activated receptor γCurrent Opinion in Genetics & Development, 1995
- Activation of immediate‐early gene expression by peroxisome proliferators in vitroMolecular Carcinogenesis, 1993
- Role of interferon γ and tumour necrosis factor α in monocyte-mediated cytostasis and cytotoxicity against a human histiocytic lymphoma cell lineCancer Immunology, Immunotherapy, 1992