New Approaches for the Treatment of Genomic Damage in End-Stage Renal Disease
- 31 January 2008
- journal article
- Published by Elsevier in Journal of Renal Nutrition
- Vol. 18 (1) , 127-133
- https://doi.org/10.1053/j.jrn.2007.10.026
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Angiotensin II-induced genomic damage in renal cells can be prevented by angiotensin II type 1 receptor blockage or radical scavengingAmerican Journal of Physiology-Renal Physiology, 2007
- Early clinical, quality-of-life, and biochemical changes of “daily hemodialysis” (6 dialyses per week)American Journal of Kidney Diseases, 2004
- Review on uremic toxins: Classification, concentration, and interindividual variabilityKidney International, 2003
- Local renin-angiotensin systems and their interactions with extra-adrenal corticosteroid productionJournal of the Renin-Angiotensin-Aldosterone System, 2002
- The elephant in uremia: Oxidant stress as a unifying concept of cardiovascular disease in uremiaKidney International, 2002
- Daily haemodialysis improves indices of protein glycationNephrology Dialysis Transplantation, 2002
- Oxidative stress in hemodialysis patients: Is NADPH oxidase complex the culprit?Kidney International, 2002
- Angiotensin II as a cardiovascular risk factorJournal of Human Hypertension, 2002
- Determination of Advanced Glycation End Products in Serum by Fluorescence Spectroscopy and Competitive ELISAcclm, 1997
- Sister chromatid exchange and chromosome abnormalities in uremic patientsCancer Genetics and Cytogenetics, 1988