Multiple death pathways in TNF-treated fibroblasts: RIP3- and RIP1-dependent and independent routes
- 4 January 2011
- journal article
- Published by Springer Nature in Cell Research
- Vol. 21 (2) , 368-371
- https://doi.org/10.1038/cr.2011.3
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- RIP3, an Energy Metabolism Regulator That Switches TNF-Induced Cell Death from Apoptosis to NecrosisScience, 2009
- Inhibition of ADP/ATP Exchange in Receptor-Interacting Protein-Mediated NecrosisMolecular and Cellular Biology, 2006
- Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injuryNature Chemical Biology, 2005
- JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen speciesGenes & Development, 2004
- Regulation of an ATG7 - beclin 1 Program of Autophagic Cell Death by Caspase-8Science, 2004
- Tumor Necrosis Factor-induced Nonapoptotic Cell Death Requires Receptor-interacting Protein-mediated Cellular Reactive Oxygen Species AccumulationJournal of Biological Chemistry, 2004
- NF- B inhibits TNF-induced accumulation of ROS that mediate prolonged MAPK activation and necrotic cell deathThe EMBO Journal, 2003
- More than one way to die: apoptosis, necrosis and reactive oxygen damageOncogene, 1999
- THE MITOCHONDRIAL DEATH/LIFE REGULATOR IN APOPTOSIS AND NECROSISAnnual Review of Physiology, 1998
- Cell membrane permeabilization and cellular collapse, followed by loss of dehydrogenase activity: Early events in tumour necrosis factor-induced cytotoxicityCytokine, 1993