Transcriptional repression by COUP-TF II is dependent on the C-terminal domain and involves the N-CoR variant, RIP13δ1
- 1 November 1997
- journal article
- Published by Elsevier in The Journal of Steroid Biochemistry and Molecular Biology
- Vol. 63 (4-6) , 165-174
- https://doi.org/10.1016/s0960-0760(97)00079-4
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Identification of a regulatory function for an orphan receptor in muscle: COUP-TF II affects the expression of themyoDgene family during myogenesisNucleic Acids Research, 1995
- A Dynamic Balance between ARP-1/COUP-TFII, EAR-3/COUP-TFI, and Retinoic Acid Receptor:Retinoid X Receptor Heterodimers Regulates Oct-3/4 Expression in Embryonal Carcinoma CellsMolecular and Cellular Biology, 1995
- An evolutionary conserved COUP-TF binding element in a neural-specific gene and COUP-TF expression patterns support a major role for COUP-TF in neural developmentMolecular Endocrinology, 1994
- COUP-TF an orphan member of the steroid/thyroid hormone receptor superfamilyTrends in Endocrinology & Metabolism, 1994
- COUP orphan receptors are negative regulators of retinoic acid response pathways.Molecular and Cellular Biology, 1992
- Chicken ovalbumin upstream promoter transcription factor (COUP-TF) dimers bind to different GGTCA response elements, allowing COUP-TF to repress hormonal induction of the vitamin D3, thyroid hormone, and retinoic acid receptors.Molecular and Cellular Biology, 1992
- Orphan receptors: in search of a unifying hypothesis for activationMolecular Endocrinology, 1992
- Repression by ARP-1 sensitizes apolipoprotein AI gene responsiveness to RXR alpha and retinoic acid.Molecular and Cellular Biology, 1992
- Dopamine Activation of an Orphan of the Steroid Receptor SuperfamilyScience, 1991
- Regulation of the Apolipoprotein AI Gene by ARP-1, a Novel Member of the Steroid Receptor SuperfamilyScience, 1991