The Stability of Tristetraprolin mRNA Is Regulated by Mitogen-activated Protein Kinase p38 and by Tristetraprolin Itself
Open Access
- 1 July 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (31) , 32393-32400
- https://doi.org/10.1074/jbc.m402059200
Abstract
No abstract availableKeywords
This publication has 62 references indexed in Scilit:
- MAPKAP Kinase 2 Phosphorylates Tristetraprolin on in Vivo Sites Including Ser178, a Site Required for 14-3-3 BindingJournal of Biological Chemistry, 2004
- p38 Mitogen-activated Protein Kinase Stabilizes mRNAs That Contain Cyclooxygenase-2 and Tumor Necrosis Factor AU-rich Elements by Inhibiting DeadenylationJournal of Biological Chemistry, 2003
- Selective Degradation of AU-Rich mRNAs Promoted by the p37 AUF1 Protein IsoformMolecular and Cellular Biology, 2003
- Transcriptional Regulation of Tristetraprolin by Transforming Growth Factor-β in Human T CellsJournal of Biological Chemistry, 2003
- Inherent Instability of Plasminogen Activator Inhibitor Type 2 mRNA Is Regulated by TristetraprolinJournal of Biological Chemistry, 2003
- AU Binding Proteins Recruit the Exosome to Degrade ARE-Containing mRNAsCell, 2001
- HuR and mRNA stabilityCellular and Molecular Life Sciences, 2001
- Regulation of tumour necrosis factor α mRNA stability by the mitogen‐activated protein kinase p38 signalling cascadeFEBS Letters, 2000
- Unraveling a cytoplasmic role for hnRNP D in the in vivo mRNA destabilization directed by the AU-rich elementGenes & Development, 1999
- Impaired On/Off Regulation of TNF Biosynthesis in Mice Lacking TNF AU-Rich ElementsImmunity, 1999