Ascorbate-induced free radical toxicity to isolated islet cells
- 31 May 1995
- journal article
- Published by Elsevier in The International Journal of Biochemistry & Cell Biology
- Vol. 27 (5) , 493-498
- https://doi.org/10.1016/1357-2725(95)00010-m
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Model Studies of the Iron-Catalysed Haber-Weiss Cycle and the Ascorbate-Driven Fenton ReactionFree Radical Research Communications, 1990
- In the absence of catalytic metals ascorbate does not autoxidize at pH 7: ascorbate as a test for catalytic metalsJournal of Biochemical and Biophysical Methods, 1988
- A site-specific mechanism for free radical induced biological damage: The essential role of redox-active transition metalsFree Radical Biology & Medicine, 1988
- Superoxide ion as a primary reductant in ascorbate-mediated ferretin iron releaseFree Radical Biology & Medicine, 1987
- Ascorbate Autoxidation in the Presence of Iron and Copper ChelatesFree Radical Research Communications, 1986
- Thioredoxin and thioredoxin reductase in pancreatic islets may participate in diabetogenic free-radical productionBiochemical and Biophysical Research Communications, 1982
- Superoxide dismutase is a prophylactic against alloxan diabetesNature, 1981
- Influence of trace metals on alloxan cytotoxicity in pancreatic isletsFEBS Letters, 1979
- Superoxide dismutase, catalase and scavengers of hydroxyl radical protect against the toxic action of alloxan on pancreatic islet cells in vitroBiochemical Journal, 1979
- Trypan Blue as a marker of plasma membrane permeability in alloxan-treated mouse islet cellsJournal of Endocrinological Investigation, 1979