Folic Acid Improves Endothelial Function in Coronary Artery Disease via Mechanisms Largely Independent of Homocysteine Lowering
- 1 January 2002
- journal article
- clinical trial
- Published by Wolters Kluwer Health in Circulation
- Vol. 105 (1) , 22-26
- https://doi.org/10.1161/hc0102.101388
Abstract
Background— Homocysteine is a risk factor for coronary artery disease (CAD), although a causal relation remains to be proven. The importance of determining direct causality rests in the fact that p...Keywords
This publication has 16 references indexed in Scilit:
- Flow-mediated dilatation following wrist and upper arm occlusion in humans: the contribution of nitric oxideClinical Science, 2001
- Vitamin Supplements and Cardiovascular Risk: Review of the Randomized Trials of Homocysteine-Lowering Vitamin SupplementsSeminars in Thrombosis and Hemostasis, 2000
- Developments in the measurement of plasma total homocysteineCurrent Opinion in Clinical Nutrition and Metabolic Care, 2000
- Endothelial dysfunction by acute hyperhomocyst(e)inaemia: restoration by folic acidClinical Science, 1999
- Reduction of Plasma Homocyst(e)ine Levels by Breakfast Cereal Fortified with Folic Acid in Patients with Coronary Heart DiseaseNew England Journal of Medicine, 1998
- Lowering blood homocysteine with folic acid based supplements: meta-analysis of randomised trialsBMJ, 1998
- A Quantitative Assessment of Plasma Homocysteine as a Risk Factor for Vascular DiseaseJAMA, 1995
- The pathogenesis of atherosclerosis: a perspective for the 1990sNature, 1993
- Stimulation of dihydrofolate reductase promoter activity by antimetabolic drugs.Proceedings of the National Academy of Sciences, 1991
- Measurement of 5-methyltetrahydrofolic acid in man by high-performance liquid chromatographyMetabolism, 1990