Correlation of 1A afferent conduction with the ataxia of fisher syndrome
- 1 May 1986
- journal article
- research article
- Published by Wiley in Muscle & Nerve
- Vol. 9 (4) , 327-332
- https://doi.org/10.1002/mus.880090408
Abstract
The pathophysiology of the ataxia in Fisher syndrome (the syndrome of acute ophthalmoplegia, ataxia, and areflexia) has been attributed to both peripheral nervous system pathology and cerebellar system dysfunction. Pathologic studies have demonstrated no consistent central nervous system abnormalities. We present a case in which abnormalities of 1a sensory conduction were found to correlate directly with the degree of ataxia, without impairment of motor or cutaneous sensory conduction. We propose that the ataxia seen in typical Fisher syndrome may be due to demyelination of 1a afferent fibers.This publication has 24 references indexed in Scilit:
- Sensory group la proximal conduction velocityMuscle & Nerve, 1984
- A new indirect method for measuring spinal conduction velocity in manElectroencephalography and Clinical Neurophysiology/Evoked Potentials Section, 1984
- Neuropathological findings in Miller Fisher syndrome.Journal of Neurology, Neurosurgery & Psychiatry, 1984
- Proposed Mechanism of Ataxia in Fisher's SyndromeArchives of Neurology, 1983
- BRAINSTEM ENCEPHALITIS AND THE SYNDROME OF MILLER FISHER A CLINICAL STUDYBrain, 1982
- Diagnostic considerations in Guillain-Barr syndromeAnnals of Neurology, 1981
- Ophthalmoplegia, Ataxia and Hyporeflexia (Fisher’s Syndrome)European Neurology, 1979
- Peripheral nerve conduction in Miller Fisher syndrome.Journal of Neurology, Neurosurgery & Psychiatry, 1977
- THE INFLAMMATORY LESION IN IDIOPATHIC POLYNEURITISMedicine, 1969
- THE LANDRY-GUILLAIN-BARR?? SYNDROMEMedicine, 1949