Critical intermediate steps in Clostridium sordellii lethal toxin-induced apoptosis
- 30 November 2007
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 363 (4) , 959-964
- https://doi.org/10.1016/j.bbrc.2007.09.073
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Continuous expression of Bcl‐xL protein during megakaryopoiesis is post‐translationally regulated by thrombopoietin‐mediated Akt activation, which prevents the cleavage of Bcl‐xLJournal of Thrombosis and Haemostasis, 2007
- PI3K-Akt pathway: Its functions and alterations in human cancerApoptosis, 2004
- A Phosphatidylserine-binding Site in the Cytosolic Fragment of Clostridium sordellii Lethal Toxin Facilitates Glucosylation of Membrane-bound Rac and Is Required for CytotoxicityJournal of Biological Chemistry, 2004
- Lethal toxin fromClostridium sordelliiinduces apoptotic cell death by disruption of mitochondrial homeostasis in HL-60 cellsCellular Microbiology, 2003
- pH-Enhanced Cytopathic Effects of Clostridium sordellii Lethal ToxinInfection and Immunity, 2001
- Rho GTPases as Targets of Bacterial Protein ToxinsBiological Chemistry, 2000
- Cellular survival: a play in three AktsGenes & Development, 1999
- The Ras-Related Protein Ral Is Monoglucosylated byClostridium sordelliiLethal ToxinBiochemical and Biophysical Research Communications, 1996
- Ras, Rap, and Rac Small GTP-binding Proteins Are Targets for Clostridium sordellii Lethal Toxin GlucosylationJournal of Biological Chemistry, 1996
- Inactivation of Ras by Clostridium sordellii Lethal Toxin-catalyzed GlucosylationJournal of Biological Chemistry, 1996