Widespread expression of α-synuclein and τ immunoreactivity in Hallervorden–Spatz syndrome with protracted clinical course
- 1 August 2000
- journal article
- case report
- Published by Elsevier in Journal of the Neurological Sciences
- Vol. 177 (1) , 48-59
- https://doi.org/10.1016/s0022-510x(00)00337-3
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Lewy body in neurodegeneration with brain iron accumulation type 1 is immunoreactive for α-synucleinNeurology, 1998
- Homozygosity mapping of Hallervorden–Spatz syndrome to chromosome 20p12.3–p13Nature Genetics, 1996
- Early Occurrence of Alzheimer's Neurofibrillary Tangles (NFT) in Developmental Brain Damage and Chronic Brain Diseases with Early Childhood OnsetNeuropathology, 1993
- Apolipoprotein E genotyping by one-stage PCRThe Lancet, 1991
- Hallervorden‐spatz syndrome: Clinical and magnetic resonance imaging correlationsAnnals of Neurology, 1988
- Hallervorden-Spatz SyndromeArchives of Neurology, 1974
- Hallervorden-Spatz disease and infantile neuroaxonal dystrophy: Ultrastructural observations, anatomical pathology and nosologyJournal of the Neurological Sciences, 1973
- Hallervorden-spatz disease and infantile neuroaxonal dystrophy: Clinical characteristics and nosological considerationsJournal of the Neurological Sciences, 1973
- Clinico-Pathological and Histochemical Studies of Hallervorden-Spatz Disease with Torsion Dystonia with Special Reference to Diagnostic Criteria of the Disease from the Clinico-Pathological ViewpointPublished by Elsevier ,1966
- Eigenartige erkrankung im extrapyramidalen system mit besonderer beteiligung des globus pallidus und der substantia nigraZeitschrift für die gesamte Neurologie und Psychiatrie, 1922