Helicobacter pylori cag-Type IV Secretion System Facilitates Corpus Colonization to Induce Precancerous Conditions in Mongolian Gerbils
- 1 May 2005
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 128 (5) , 1229-1242
- https://doi.org/10.1053/j.gastro.2005.02.064
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- TheHelicobacter pyloriplasticity region locusjhp0947âjhp0949is associated with duodenal ulcer disease and interleukin-12 production in monocyte cellsFEMS Immunology & Medical Microbiology, 2004
- Mice deficient in protein tyrosine phosphatase receptor type Z are resistant to gastric ulcer induction by VacA of Helicobacter pyloriNature Genetics, 2003
- Colonization of C57BL/6J and BALB/c Wild-Type and Knockout Mice withHelicobacter pylori: Effect of Vaccination and Implications for Innate and Acquired ImmunityInfection and Immunity, 2003
- Helicobacter pylori and gastrointestinal tract adenocarcinomasNature Reviews Cancer, 2002
- Interleukin-1 polymorphisms associated with increased risk of gastric cancerNature, 2000
- Translocation of Helicobacter pylori CagA into Gastric Epithelial Cells by Type IV SecretionScience, 2000
- EGF receptor activation stimulates endogenous gastrin gene expression in canine G cells and human gastric cell cultures.Journal of Clinical Investigation, 1997
- Mosaicism in Vacuolating Cytotoxin Alleles of Helicobacter pyloriJournal of Biological Chemistry, 1995
- CagA/cytotoxic strains of Helicobacter pylori and interleukin-8 in gastric epithelial cell lines.Journal of Clinical Pathology, 1994
- Acid secretion and sensitivity to gastrin in patients with duodenal ulcer: effect of eradication of Helicobacter pylori.Gut, 1993