Comparative analysis of the methylation status of the 5′ flanking region of Ha-ras in B6C3F1, C3H/He and C57BL/6 mouse liver
- 10 December 1993
- journal article
- research article
- Published by Elsevier in Cancer Letters
- Vol. 75 (2) , 129-136
- https://doi.org/10.1016/0304-3835(93)90197-h
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Hypomethylation of DNA: A Possible Nongenotoxic Mechanism Underlying the Role of Cell Proliferation in CarcinogenesisEnvironmental Health Perspectives, 1993
- Predisposition to Neoplastic Transformation Caused by Gene Replacement of H- ras 1Science, 1993
- Biological aspects of cytosine methylation in eukaryotic cellsCellular and Molecular Life Sciences, 1991
- Mouse Liver Carcinogenesis: Mechanisms and Relevance*1Fundamental and Applied Toxicology, 1991
- DNA methylation inhibits transcription indirectly via a methyl-CpG binding proteinPublished by Elsevier ,1991
- CpG methylation inhibits proenkephalin gene expression and binding of the transcription factor AP-2Nucleic Acids Research, 1990
- A point mutation in the last intron responsible for increased expression and transforming activity of the c-Ha-ras oncogeneNature, 1988
- Isolation and characterization of the 5′ flanking region of the mouse C‐harvey‐ras geneMolecular Carcinogenesis, 1988
- CpG-rich islands and the function of DNA methylationNature, 1986
- Tumorigenic transformation of mammalian cells induced by a normal human gene homologous to the oncogene of Harvey murine sarcoma virusNature, 1982