A herpes simplex virus ribonucleotide reductase deletion mutant is defective for productive acute and reactivatable latent infections of mice and for replication in mouse cells
- 1 November 1989
- Vol. 173 (1) , 276-283
- https://doi.org/10.1016/0042-6822(89)90244-4
Abstract
No abstract availableThis publication has 47 references indexed in Scilit:
- Herpes simplex virus ribonucleotide reductase mutants are hypersensitive to acyclovirAntimicrobial Agents and Chemotherapy, 1989
- Ribonucleotide Reductase Encoded by Herpes Simplex Virus Is a Determinant of the Pathogenicity of the Virus in Mice and a Valid Antiviral TargetJournal of General Virology, 1988
- Factor(s) present in herpes simplex virus type 1-infected cells can compensate for the loss of the large subunit of the viral ribonucleotide reductase: characterization of an ICP6 deletion mutantVirology, 1988
- Specific inhibition of herpesvirus ribonucleotide reductase by a nonapeptide derived from the carboxy terminus of subunit 2Nature, 1986
- Specific inhibition of herpesvirus ribonucleotide reductase by synthetic peptidesNature, 1986
- The Ribonucleotide Reductase Induced by Herpes Simplex Virus Type 1 Involves Minimally a Complex of Two Polypeptides (136K and 38K)Journal of General Virology, 1985
- Ribonucleotide Reductase Induced by Herpes Simplex Virus has a Virus-specified ConstituentJournal of General Virology, 1983
- The pathogenicity of thymidine kinase-deficient mutants of herpes simplex virus in miceEpidemiology and Infection, 1978
- Induction of Both Thymidine and Deoxycytidine Kinase Activity by Herpes VirusesJournal of General Virology, 1974
- Mutant strains of herpes simplex deficient in thymidine kinase-inducing activityVirology, 1964