Intraneuronal Alzheimer Aβ42 Accumulates in Multivesicular Bodies and Is Associated with Synaptic Pathology
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Open Access
- 1 November 2002
- journal article
- research article
- Published by Elsevier
- Vol. 161 (5) , 1869-1879
- https://doi.org/10.1016/s0002-9440(10)64463-x
Abstract
No abstract availableKeywords
This publication has 66 references indexed in Scilit:
- A Cargo Receptor Mystery APP arently Solved?Science, 2002
- Selective cytotoxicity of intracellular amyloid β peptide1–42 through p53 and Bax in cultured primary human neuronsThe Journal of cell biology, 2002
- The 'Arctic' APP mutation (E693G) causes Alzheimer's disease by enhanced Aβ protofibril formationNature Neuroscience, 2001
- Accumulation and Aggregation of Amyloid β-Protein in Late Endosomes of Niemann-Pick Type C CellsJournal of Biological Chemistry, 2001
- Aβ42-positive non-pyramidal neurons around amyloid plaques in Alzheimer's diseaseThe Lancet, 2000
- Amyloid ? protein is internalized selectively by hippocampal field CA1 and causes neurons to accumulate amyloidogenic carboxyterminal fragments of the amyloid precursor proteinJournal of Comparative Neurology, 1998
- KIFC2 Is a Novel Neuron-Specific C-Terminal Type Kinesin Superfamily Motor for Dendritic Transport of Multivesicular Body-Like OrganellesNeuron, 1997
- Correlative Memory Deficits, Aβ Elevation, and Amyloid Plaques in Transgenic MiceScience, 1996
- Intracellular Accumulation of β-Amyloid in Cells Expressing the Swedish Mutant Amyloid Precursor ProteinJournal of Biological Chemistry, 1995
- β-amyloid precursor protein-deficient mice show reactive gliosis and decreased locomotor activityCell, 1995