APP Processing and Synaptic Plasticity in Presenilin-1 Conditional Knockout Mice
- 1 September 2001
- Vol. 31 (5) , 713-726
- https://doi.org/10.1016/s0896-6273(01)00417-2
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health
- Alzheimer's Association
This publication has 58 references indexed in Scilit:
- X11α and X11β Interact with Presenilin-1 via Their PDZ DomainsMolecular and Cellular Neuroscience, 2000
- Neuron‐Specific Phosphorylation of Alzheimer's β‐Amyloid Precursor Protein by Cyclin‐Dependent Kinase 5Journal of Neurochemistry, 2000
- Synaptic Assembly of the Brain in the Absence of Neurotransmitter SecretionScience, 2000
- Enhanced Synaptic Potentiation in Transgenic Mice Expressing presenilin 1 Familial Alzheimer's Disease Mutation Is Normalized with a BenzodiazepineNeurobiology of Disease, 2000
- Synaptic Transmission and Hippocampal Long-Term Potentiation in Transgenic Mice Expressing FAD-Linked Presenilin 1Neurobiology of Disease, 1999
- Control of Memory Formation Through Regulated Expression of a CaMKII TransgeneScience, 1996
- Accumulation of β-Amyloid fibrils in pancreas of transgenic miceNeurobiology of Aging, 1996
- A single protocol to detect transcripts of various types and expression levels in neural tissue and cultured cells: in situ hybridization using digoxigenin-labelled cRNA probesHistochemistry and Cell Biology, 1993
- The carboxy terminus of the .beta. amyloid protein is critical for the seeding of amyloid formation: Implications for the pathogenesis of Alzheimer's diseaseBiochemistry, 1993
- Amyloid β-peptide is produced by cultured cells during normal metabolismNature, 1992