Amyloid β Peptide Formation in Cell-free Preparations

This publication has 24 references indexed in Scilit:

• Role of the β‐amyloid protein in Alzheimer's disease
  The FASEB Journal, 1995
• Excessive production of amyloid beta-protein by peripheral cells of symptomatic and presymptomatic patients carrying the Swedish familial Alzheimer disease mutation.
  Proceedings of the National Academy of Sciences, 1994
• An Increased Percentage of Long Amyloid β Protein Secreted by Familial Amyloid β Protein Precursor (βApp ₇₁₇ ) Mutants
  Science, 1994
• beta-Amyloid Formation as a Potential Therapeutic Target for Alzheimer's Disease
  Annual Review of Pharmacology and Toxicology, 1994
• Release of Excess Amyloid β Protein from a Mutant Amyloid β Protein Precursor
  Science, 1993
• Mutation of the β-amyloid precursor protein in familial Alzheimer's disease increases β-protein production
  Nature, 1992
• Production of the Alzheimer Amyloid β Protein by Normal Proteolytic Processing
  Science, 1992
• Isolation and quantification of soluble Alzheimer's β-peptide from biological fluids
  Nature, 1992
• Amyloid β-peptide is produced by cultured cells during normal metabolism
  Nature, 1992
• Presenile dementia and cerebral haemorrhage linked to a mutation at codon 692 of the β–amyloid precursor protein gene
  Nature Genetics, 1992