Cockayne syndrome exhibits dysregulation of p21 and other gene products that may be independent of transcription-coupled repair

This publication has 61 references indexed in Scilit:

• DNA damage in transcribed genes induces apoptosis via the JNK pathway and the JNK-phosphatase MKP-1
  Oncogene, 2005
• Tau Suppression in a Neurodegenerative Mouse Model Improves Memory Function
  Science, 2005
• Severe growth retardation and short life span of double-mutant mice lacking Xpa and exon 15 of Xpg
  DNA Repair, 2005
• Oxidative nucleotide damage and superoxide dismutase expression in the brains of xeroderma pigmentosum group A and Cockayne syndrome
  Brain & Development, 2005
• Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death
  Nature, 2004
• Proteolytic Stress Causes Heat Shock Protein Induction, Tau Ubiquitination, and the Recruitment of Ubiquitin to Tau-Positive Aggregates in Oligodendrocytes in Culture
  Journal of Neuroscience, 2004
• UV Irradiation Triggers Ubiquitin-Dependent Degradation of p21WAF1 to Promote DNA Repair
  Cell, 2003
• A proteomics approach to understanding protein ubiquitination
  Nature Biotechnology, 2003
• The Comparative Proteomics of Ubiquitination in Mouse
  Genome Research, 2003
• Identification of the regulatory region required for ubiquitination of the cyclin kinase inhibitor, p21
  Biochemical and Biophysical Research Communications, 2002