In Vivo 2-Deoxyglucose Administration Preserves Glucose and Glutamate Transport and Mitochondrial Function in Cortical Synaptic Terminals after Exposure to Amyloid β-Peptide and Iron: Evidence for a Stress Response

This publication has 52 references indexed in Scilit:

• Dietary restriction and 2-deoxyglucose administration improve behavioral outcome and reduce degeneration of dopaminergic neurons in models of Parkinson's disease
  Journal of Neuroscience Research, 1999
• Altered Calcium Homeostasis and Mitochondrial Dysfunction in Cortical Synaptic Compartments of Presenilin‐1 Mutant Mice
  Journal of Neurochemistry, 1999
• Food restriction reduces brain damage and improves behavioral outcome following excitotoxic and metabolic insults
  Annals of Neurology, 1999
• Ischemic Preconditioning and Brain Tolerance: Temporal Histological and Functional Outcomes, Protein Synthesis Requirement, and Interleukin-1 Receptor Antagonist and Early Gene Expression
  Stroke, 1998
• In VivoHeat Shock Protects Rat Myocardial Mitochondria
  Biochemical and Biophysical Research Communications, 1998
• 4-hydroxynonenal, a lipid peroxidation product, impairs glutamate transport in cortical astrocytes
  Glia, 1998
• Structural Correlates of Cognition in Dementia: Quantification and Assessment of Synapse Change
  Neurodegeneration, 1996
• Mechanisms of Neuronal Death in Alzheimer's Disease
  Brain Pathology, 1996
• Stress Proteins and Tolerance to Focal Cerebral Ischemia
  Journal of Cerebral Blood Flow & Metabolism, 1996
• The degree of protection provided to neuronal cells by a pre-conditioning stress correlates with the amount of heat shock protein 70 it induces and not with the similarity of the subsequent stress
  Neuroscience Letters, 1995