Loss of Presenilin Function Causes Impairments of Memory and Synaptic Plasticity Followed by Age-Dependent Neurodegeneration

This publication has 53 references indexed in Scilit:

• A CBP Binding Transcriptional Repressor Produced by the PS1/ϵ-Cleavage of N-Cadherin Is Inhibited by PS1 FAD Mutations
  Cell, 2003
• Presenilin 1 is involved in maturation and trafficking of N‐cadherin to the plasma membrane
  Journal of Neuroscience Research, 2003
• Disruption of Dendritic Translation of CaMKIIα Impairs Stabilization of Synaptic Plasticity and Memory Consolidation
  Neuron, 2002
• A Presenilin 1 Mutation Associated with Familial Frontotemporal Dementia Inhibits γ-Secretase Cleavage of APP and Notch
  Neurobiology of Disease, 2002
• The molecular basis of CaMKII function in synaptic and behavioural memory
  Nature Reviews Neuroscience, 2002
• APP Processing and Synaptic Plasticity in Presenilin-1 Conditional Knockout Mice
  Neuron, 2001
• A Transcriptively Active Complex of APP with Fe65 and Histone Acetyltransferase Tip60
  Science, 2001
• The Presenilin 1 C92S Mutation Increases Aβ 42 Production
  Biochemical and Biophysical Research Communications, 2000
• CREB AND MEMORY
  Annual Review of Neuroscience, 1998
• A Presenilin-1 Truncating Mutation Is Present in Two Cases with Autopsy-Confirmed Early-Onset Alzheimer Disease
  American Journal of Human Genetics, 1998