Tyrosine-Phosphorylation-Dependent Translocation of the SLAT Protein to the Immunological Synapse Is Required for NFAT Transcription Factor Activation

This publication has 43 references indexed in Scilit:

• An immune escape screen reveals Cdc42 as regulator of cancer susceptibility to lymphocyte-mediated tumor suppression
  Blood, 2008
• Structure-Function Analysis of the WIP Role in T Cell Receptor-stimulated NFAT Activation
  Published by Elsevier ,2007
• SLAT regulates Th1 and Th2 inflammatory responses by controlling Ca2+/NFAT signaling
  Journal of Clinical Investigation, 2007
• TCR/CD28-Stimulated Actin Dynamics Are Required for NFAT1-Mediated Transcription of c-rel Leading to CD28 Response Element Activation
  The Journal of Immunology, 2007
• Protein transduction as a means of effective manipulation of Cdc42 activity in primary T cells
  Journal of Immunological Methods, 2007
• Defective Th1 Cytokine Gene Transcription in CD4+ and CD8+ T Cells from Wiskott-Aldrich Syndrome Patients
  The Journal of Immunology, 2006
• Molecular identification of the CRAC channel by altered ion selectivity in a mutant of Orai
  Nature, 2006
• Role of the Guanosine Triphosphatase Rac2 in T Helper 1 Cell Differentiation
  Science, 2000
• Vav modulation of the Ras / MEK / ERK signaling pathway plays a role in NFAT activation and CD69 up-regulation
  European Journal of Immunology, 2000
• Plat-E: an efficient and stable system for transient packaging of retroviruses
  Gene Therapy, 2000