A review of evidence for GABergic predominance/glutamatergic deficit as a common etiological factor in both schizophrenia and affective psychoses: More support for a continuum hypothesis of ?functional? psychosis
- 1 October 1991
- journal article
- review article
- Published by Springer Nature in Neurochemical Research
- Vol. 16 (10) , 1099-1111
- https://doi.org/10.1007/bf00966587
Abstract
No abstract availableKeywords
This publication has 142 references indexed in Scilit:
- Convulsant potencies of tetrazoles are highly correlated with actions on GABA/benzodiazepine/picrotoxin receptor complexes in brainPublished by Elsevier ,2002
- Enhancement by chlordiazepoxide of catalepsy induced in rats by intravenous or intrapallidal injections of enantiomeric cannabinoidsNeuropharmacology, 1991
- Anatomical Abnormalities in the Brains of Monozygotic Twins Discordant for SchizophreniaNew England Journal of Medicine, 1990
- Non-competitive inhibition of GABA currents by phenothiazines in cultured chick spinal cord and rat hippocampal neuronsNeuroscience Letters, 1988
- Neurochemistry of GABAergic System in Cerebral Cortex Chronically Exposed to Bromide In VivoJournal of Neurochemistry, 1987
- Iontophoretic studies on rat hippocampus with some novel GABA antagonistsLife Sciences, 1986
- A study of the actions of bromide on frog sympathetic ganglionNeuropharmacology, 1982
- Pharmacology of excitatory amino acid receptors mediating the stimulation of rat cerebellar cyclic GMP leveleLife Sciences, 1980
- EEG Profile Studies of Clozapine in Volunteers and Psychiatric PatientsPharmacopsychiatry, 1979
- Bicyclic phosphorus esters increase the cyclic GMP level in rat cerebellumNature, 1977