Early activation of the mitochondrial apoptotic pathway in Vesicular Stomatitis Virus-infected cells
- 30 April 2005
- journal article
- Published by Elsevier in Virus Research
- Vol. 109 (1) , 65-69
- https://doi.org/10.1016/j.virusres.2004.10.007
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Requirement of caspase-3 for efficient apoptosis induction and caspase-7 activation but not viral replication or cell rounding in cells infected with vesicular stomatitis virusHuman Immunology, 2002
- Vesicular stomatitis virus induces apoptosis at early stages in the viral cycle and does not depend on virus replicationVirus Research, 2002
- Matrix Protein Mutations Contribute to Inefficient Induction of Apoptosis Leading to Persistent Infection of Human Neural Cells by Vesicular Stomatitis VirusVirology, 2002
- Caspase-3-like proteases are activated by infection but are not required for replication of vesicular stomatitis virusVirus Research, 2001
- Oncolytic Activity of Vesicular Stomatitis Virus Is Effective against Tumors Exhibiting Aberrant p53, Ras, or Myc Function and Involves the Induction of ApoptosisJournal of Virology, 2001
- Apoptosis‐inducing factor (AIF): a ubiquitous mitochondrial oxidoreductase involved in apoptosisFEBS Letters, 2000
- Alpha/Beta Interferons Potentiate Virus-Induced Apoptosis through Activation of the FADD/Caspase-8 Death Signaling PathwayJournal of Virology, 2000
- BCL-2 family members and the mitochondria in apoptosisGenes & Development, 1999
- Alphaviruses induce apoptosis in Bcl-2-overexpressing cells: evidence for a caspase-mediated, proteolytic inactivation of Bcl-2The EMBO Journal, 1998
- Modulation of cell death by Bcl-x L through caspase interactionProceedings of the National Academy of Sciences, 1998