Atherosclerosis or lipoprotein-induced endothelial dysfunction. Potential mechanisms underlying reduction in EDRF/nitric oxide activity.

This publication has 53 references indexed in Scilit:

• Analysis of the monocyte chemotactic response to lysophosphatidylcholine: role of lysophospholipase C
  Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1991
• Thromboxane A2 receptor antagonists inhibit endothelium-dependent contractions.
  Hypertension, 1990
• Prostaglandin H2 may be the endothelium-derived contracting factor released by acetylcholine in the aorta of the rat.
  Hypertension, 1990
• Lysophosphatidylcholine: Essential role in the inhibition of endothelium-dependent vasorelaxation by oxidized low density lipoprotein
  Biochemical and Biophysical Research Communications, 1990
• Regulation of the mevalonate pathway
  Nature, 1990
• Aging of rat heart myocytes disrupts muscarinic receptor coupling that leads to inhibition of cAMP accumulation and alters the pathway of muscarinic-stimulated phosphoinositide hydrolysis
  Biochemistry, 1989
• Mediators produced by the endothelial cell.
  Hypertension, 1988
• Role of superoxide in endothelial-cell modification of low-density lipoproteins
  Biochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1988
• The Pathogenesis of Atherosclerosis — An Update
  New England Journal of Medicine, 1986
• Protein kinase C interferes with N_i‐mediated inhibition of human platelet adenylate cyclase
  FEBS Letters, 1985