Abstract
SO2 supplied at constant (square wave) or variable (triangular wave) concentrations affected development of lesions caused by X. phaseoli by increasing the latent period (time for symptom expression) and suppressing lesion expansion. Similar effects were found with both patterns of exposure. Timing of exposures relative to inoculation was important and generally was most effective when the exposure and inoculation periods were close together. The time between repeated triangular wave SO2 exposures also affected lesion development and indicated a decay of inhibition if exposures were separated by > 1 day.

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