A Novel Splice Variant of the Cell Death-promoting Protein BAX
Open Access
- 1 May 1998
- journal article
- Published by Elsevier
- Vol. 273 (19) , 11930-11936
- https://doi.org/10.1074/jbc.273.19.11930
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- The Bcl-2 family of proteins, and the regulation of neuronal survivalTrends in Neurosciences, 1995
- Mapping of the human BAX gene to chromosome 19q13.3–q13.4 and isolation of a novel alternatively spliced transcript, BAXδGenomics, 1995
- The protein bcl-2 alpha does not require membrane attachment, but two conserved domains to suppress apoptosis.The Journal of cell biology, 1994
- Bcl-2 and the regulation of programmed cell deathThe Journal of cell biology, 1994
- Bcl-2 functions in an antioxidant pathway to prevent apoptosisCell, 1993
- Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell deathCell, 1993
- bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell deathCell, 1993
- Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell deathNature, 1990
- A novel genetic system to detect protein–protein interactionsNature, 1989
- Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cellsNature, 1988