GM-CSF RESCUES TF-1 CELLS FROM GROWTH FACTOR WITHDRAWAL-INDUCED, BUT NOT DIFFERENTIATION-INDUCED APOPTOSIS: THE ROLE OF BCL-2 AND MCL-1
- 30 November 1999
- Vol. 11 (11) , 849-855
- https://doi.org/10.1006/cyto.1999.0514
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Chapter 9 The End of the (Cell) Line: Methods for the Study of Apoptosis in VitroPublished by Elsevier ,2008
- Terminal megakaryocytic differentiation of TF-1 cells is induced by phorbol esters and thrombopoietin and is blocked by expression of PML/RARα fusion proteinLeukemia, 1998
- Induction of BCL2 family member MCL1 as an early response to DNA damageOncogene, 1997
- Role for Bcl-xLin the Regulation of Apoptosis by EGF and TGFβ1 in c-myc Overexpressing Mammary Epithelial CellsBiochemical and Biophysical Research Communications, 1996
- Life, death, and the pursuit of apoptosis.Genes & Development, 1996
- Multiple Bcl-2 family members demonstrate selective dimerizations with Bax.Proceedings of the National Academy of Sciences, 1995
- Apoptosis in the Pathogenesis and Treatment of DiseaseScience, 1995
- Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell deathCell, 1993
- Establishment and characterization of a unique human cell line that proliferates dependently on GM‐CSF, IL‐3, or erythropoietinJournal of Cellular Physiology, 1989
- Cell Death: The Significance of ApoptosisPublished by Elsevier ,1980