ICOS Ligand Costimulation Is Required for T-Cell Encephalitogenicity

This publication has 30 references indexed in Scilit:

• Inducible Costimulator Protein (Icos) Controls T Helper Cell Subset Polarization after Virus and Parasite Infection
  The Journal of Experimental Medicine, 2000
• Differential Cytokine and Chemokine Production Characterizes Experimental Autoimmune Meningitis and Experimental Autoimmune Encephalomyelitis
  Clinical Immunology, 2000
• B7h, a Novel Costimulatory Homolog of B7.1 and B7.2, Is Induced by TNFα
  Immunity, 1999
• Decreased dependence of myelin basic protein-reactive T cells on CD28-mediated costimulation in multiple sclerosis patients. A marker of activated/memory T cells.
  Journal of Clinical Investigation, 1998
• Myelin Basic Protein–specific T Helper 2 (Th2) Cells Cause Experimental Autoimmune Encephalomyelitis in Immunodeficient Hosts Rather than Protect Them from the Disease
  The Journal of Experimental Medicine, 1997
• Distinct roles for B7-1 (CD-80) and B7-2 (CD-86) in the initiation of experimental allergic encephalomyelitis.
  Journal of Clinical Investigation, 1995
• Animal models of human disease Experimental autoimmune encephalomyelitis in rodents as a model for human demyelinating disease
  Clinical Immunology and Immunopathology, 1995
• B7-1 and B7-2 costimulatory molecules activate differentially the Th1/Th2 developmental pathways: Application to autoimmune disease therapy
  Cell, 1995
• High incidence of spontaneous autoimmune encephalomyelitis in immunodeficient anti-myelin basic protein T cell receptor transgenic mice
  Cell, 1994
• Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
  Cell, 1993