Modulation of MK-801-elicited mouse popping behavior by galantamine is complex and dose-dependent
- 1 September 2003
- journal article
- Published by Elsevier in Life Sciences
- Vol. 73 (18) , 2355-2361
- https://doi.org/10.1016/s0024-3205(03)00642-8
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- Allosteric sensitization of nicotinic receptors by galantamine, a new treatment strategy for Alzheimer’s diseaseBiological Psychiatry, 2001
- A Revised Excitotoxic Hypothesis of Schizophrenia: Therapeutic ImplicationsClinical Neuropharmacology, 2001
- Decreased protein level of nicotinic receptor α7 subunit in the frontal cortex from schizophrenic brainNeuroReport, 1999
- The Neuropsychopharmacology of Phencyclidine From NMDA Receptor Hypofunction to the Dopamine Hypothesis of SchizophreniaNeuropsychopharmacology, 1999
- Chapter 28 The glutamate synapse in neuropsychiatric disorders: Focus on schizophrenia and Alzheimer's diseasePublished by Elsevier ,1998
- Choline is a Selective Agonist of α7 Nicotinic Acetylcholine Receptors in the Rat Brain NeuronsEuropean Journal of Neuroscience, 1997
- The Glutamatergic Dysfunction Hypothesis for SchizophreniaHarvard Review of Psychiatry, 1996
- Computerized Measurement of MK-801-Elicited Popping and Hyperactivity in MiceClinical Neuropharmacology, 1995
- Evidence in postmortem brain tissue for decreased numbers of hippocampal nicotinic receptors in schizophreniaBiological Psychiatry, 1995
- A “Glutamatergic Hypothesis” of SchizophreniaClinical Neuropharmacology, 1989