Interaction at clinical level between erythrocyte acid phosphatase and adenosine deaminase genetic polymorphisms
- 1 June 1989
- journal article
- research article
- Published by Springer Nature in Human Genetics
- Vol. 82 (3) , 213-215
- https://doi.org/10.1007/bf00291156
Abstract
The effects of ACP1 phenotype on birth weight, neonatal jaundice, and obesity in children are dependent on ADA genotype. These phenomena may represent a clinical counterpart of the in vitro biochemical interactions between the two systems recently observed by our group.Keywords
This publication has 19 references indexed in Scilit:
- Foetal macrosomia and erythrocyte acid phosphatase (ACP1) polymorphism in diabetic and normal pregnancyEarly Human Development, 1988
- Serum haptoglobin appearance during neonatal period is associated with acid phosphatase (ACP1) phenotypeEarly Human Development, 1985
- NEONATAL JAUNDICE AND ERYTHROCYTE-ACID-PHOSPHATASE PHENOTYPEThe Lancet, 1976
- Comparative activity of red cell adenosine deaminase allelic formsNature, 1974
- Activity of the ‘red cell’ acid phosphatase locus in other tissuesAnnals of Human Genetics, 1973
- Erythrocyte acid phosphatase polymorphism and haemolysis.Journal of Medical Genetics, 1972
- Adenosine deaminase isozymes in human tissuesAnnals of Human Genetics, 1971
- Favism: Association with Erythrocyte Acid Phosphatase PhenotypeScience, 1971
- Adenosine deaminase polymorphism in manAnnals of Human Genetics, 1968
- Red Cell Acid Phosphatase Variants: A New Human PolymorphismNature, 1963