The effects of daily cyclophosphamide administration on the development and extent of primary experimental interstitial nephritis in rats
- 1 March 1986
- journal article
- research article
- Published by Elsevier in Kidney International
- Vol. 29 (3) , 635-640
- https://doi.org/10.1038/ki.1986.46
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Inhibitory role of dietary protein restriction on the development and expression of immune-mediated antitubular basement membrane-induced tubulointerstitial nephritis in rats.Journal of Clinical Investigation, 1985
- Antiidiotypic immunity in interstitial nephritis. II. Rats developing anti-tubular basement membrane disease fail to make an antiidiotypic regulatory response: the modulatory role of an RT7.1+, OX8- suppressor T cell mechanism.The Journal of Experimental Medicine, 1984
- Characterization of inflammatory cells in autoimmune tubulointerstitial nephritis in ratsKidney International, 1983
- Suppression of interstitial nephritis by auto-anti-idiotypic immunity.The Journal of Experimental Medicine, 1982
- Differential sensitivity to cyclophosphamide of helper T cells for humoral responses and suppressor T cells for delayed‐type hypersensitivityEuropean Journal of Immunology, 1980
- The Clinical Pharmacology of Antineoplastic AgentsNew England Journal of Medicine, 1975
- Cyclophosphamide: Use in PracticeAnnals of Internal Medicine, 1974
- Therapeutic studies in NZB/W mice. II. Relative efficacy of azathioprine, cyclophosphamide and methylprednisoloneArthritis & Rheumatism, 1972
- Therapeutic studies in NZB/W mice. I. Synergy of azathioprine, cyclophosphamide and methylprednisolone in combinationArthritis & Rheumatism, 1972
- Activation of cyclophosphamide in man and animalsCancer, 1971