Chloroquine inhibits the production of a mononuclear cell factor by inhibition of lectin binding

Abstract
The effect of chloroquine (CQ) on the ability of normal human peripheral blood monocytes to produce a soluble mononuclear cell factor (MCF) capable of stimulating prostaglandins from adherent rheumatoid synovial cells was studied. CQ inhibited concanavalin A (Con A)‐ and bacterial peptidoglycan‐induced MCF activity, but not colchicine‐induced activity, in a dose‐dependent manner. After incubation with CQ for 20 minutes, 3H Con A binding was reduced by 50%, and metabolism of pre‐bound 3H Con A was inhibited. These data suggest that CQ can inhibit MCF production by inhibition of lectin binding, perhaps by inhibition of receptor recycling.

This publication has 30 references indexed in Scilit: