The Landry–Guillain–Barré Syndrome

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Abstract
SINCE the original descriptions of the Landry–Guillain–Barré syndrome, first by Landry,1 in 1859, as a brief illness characterized by rapidly spreading paralysis with minimal sensory symptoms and little or no increase of the protein in the spinal fluid, and then by Guillain, Barré and Strohl,2 in 1916, and by Guillain,3 in 1936, as a syndrome of paralysis of the peripheral muscles that pursued a benign course ending in recovery and characterized by an increase of protein in the spinal fluid to 1 to 2 gm. per 100 ml., with no pleocytosis, great debate and controversy have revolved around the criteria . . .

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